Analysis of Cardiac Development: from Embryo to Old Age (Annals of the New York Academy of Sciences)
By: Amir Landesberg (editor), Rafael Beyar (editor)Paperback
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This volume, the result of three days of interactive sessions amongworld leaders in the cardiac sciences, summarizes the mostup-to-date information about development and cardiogenesissignaling in cell-based therapy, as well as developmental aspectsof the formation of the embryonic heart, including the effect ofmechanical load on differentiation.
Other topics covered include: signaling and repair strategies,cell and gene therapy for the treatment of postmyocardialinfarction, signaling, vascularization methods in engineeringembryonic cardiac tissue, and molecular methods to improve survivalof human embryonic stem cell derived cardiomyocytes;developmental and evolutional cardiology; novel strategies fortreatment of atrial fibrillation and channel molecular physiologyin remodeling and hypertrophy; multiscale modeling for metabolismand flows, including force development, mechanics of cardiaccontraction, and ATP supply and demand aspects; aging,interactions, and interference aspects includefibroblast-myocyte-capillary communications, nonuniformities incontraction, calcium channels as oxygen sensors, and epigenetics ofheart failure; and macroscale phenomena and clinical aspects,including various clinical aspects of modern cardiology such asnavigation methods for cardiac interventions and control of cardiacfunction by changes in energetic demand. NOTE: Annals volumes are available for sale as individual booksor as a journal. For information on institutional journalsubscriptions, please visit www.blackwellpublishing.com/nyas.
ACADEMY MEMBERS: Please contact the New York Academy of Sciencesdirectly to place your order (www.nyas.org). Members of the NewYork Academy of Science receive full-text access to the Annalsonline and discounts on print volumes. Please visit http://www.nyas.org/MemberCenter/Join.aspxfor more information about becoming a member.
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- ID: 9781573317474
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