Oral Medicine and Pathology at a Glance (At a Glance (Dentistry))

Oral Medicine and Pathology at a Glance (At a Glance (Dentistry))

By: Jose V. Bagan (author), Oslei Paes de Almeida (author), Pedro Diz Dios (author), Adalberto Mosqueda Taylor (author), Crispian Scully (author)Paperback

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Oral Medicine and Pathology At A Glance is a title in the highly popular at a Glance series. It provides a concise and accessible introduction and revision aid. Following the familiar, easy-to-use at a Glance format, each topic is presented as a double-page spread with key facts accompanied by clear diagrams, clinical photographs, and useful quick-reference tables, encapsulating essential information.

Systematically organized and succinctly delivered, Oral Medicine and Pathology At A Glance covers: * The most important conditions, by frequency and severity, in oral medicine and pathology * Commonly encountered oral lesions and pathologies * Oral manifestations of common systemic diseases * Differential diagnosis * Clinical management of these conditions

Oral Medicine and Pathology At A Glance is the ideal companion for all students of dentistry and recently qualified clinicians. In addition, through its focus on oral health care provision in general practice, the text will provide valuable insight for general dental practitioners wanting to update their knowledge of oral medicine and pathology, dental nurses, hygienists and therapists.

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About Author

Crispian Scully is Professor of Oral Medicine, Pathology andMicrobiology at University of London, Director (Special Projects)at the UCL-Eastman Dental Institute, and Professor of Special CareDentistry. He also serves as Chair of Division of MaxillofacialDiagnostic, Medical and Surgical Sciences and is President-elect ofthe International Academy of Oral Oncology (IAOO). He is VisitingProfessor at the Universities of Bristol, Edinburgh and Helsinki.

Oslei Paes de Almeida is Professor in the Department ofOral Diagnosis and Pathology, Dental School of Piracicaba,University of Campinas, Sao Paulo, Brazil.

Jose Vicente Bagan is Professor of Oral Medicine atValencia University, Department of Stomatology, University GeneralHospital, Valencia, Spain.

Pedro Diz Dios is Senior Lecturer in Special NeedsDentistry and Head of Special Needs Dentistry Section within theSchool of Medicine and Dentistry at Santiago de CompostelaUniversity, Spain. Additionally he is Honorary Visiting Professorat UCL-Eastman Dental Institute, University College of London(UK).

Adalberto Mosqueda Taylor is Professor of Oral Pathologyand Medicine at the Health Care Department, Universidad AutonomaMetropolitana Xochimilco, and Honorary Professor at the NationalInstitute of Cancerology in Mexico City.



1 Examination of extraoral tissues.

Head and neck.

Cranial nerves.


2 Examination of mouth, jaws, temporomandibular region andsalivary glands.



Temporomandibular joint (TMJ).

Salivary glands.

3 Investigations: Histopathology.

Mucosal biopsy.

Brush biopsy.

Labial salivary gland biopsy.

4 Investigations: Microbiology.

5 Investigations: Imaging.

6 Investigations: Blood tests.

Referring a patient for specialist opinion.

7 Anatomical variants and developmental anomalies.

Fordyce spots ("Fordyce granules").

Fissured tongue (scrotal or plicated tongue).

Stafne cyst or bone cavity.

Torus palatinus.

Torus mandibularis.


8 Blisters.

Angina bullosa hemorrhagica (localized oral purpura; traumaticoral hemophlyctenosis).

9 Blisters, infections: Herpes simplex virus.

Herpes simplex.

Recurrent herpes labialis.

Recurrent intraoral herpes.

10 Blisters infections: Varicella zoster virus.

Chickenpox (varicella).

Zoster (shingles).

11 Blisters, skin diseases: Pemphigus.


12 Blisters, skin diseases: Pemphigoid.

13 Pigmented lesions.

Superficial discoloration.

Hairy tongue (black hairy tongue; lingua villosa nigra).

14 Pigmented lesions: Ethnic pigmentation andtattoos.

Ethnic pigmentation.

Foreign body tattoos.

15 Pigmented lesions: Melanotic macule.

16 Pigmented lesions: Nevus and others.

Adenocorticotrophic hormone effects (ACTH).

17 Pigmented lesions: Malignant melanoma.

18 Red and purple lesions.


19 Red and purple lesions: Desquamative gingivitis,mucositis.

Desquamative gingivitis.


20 Red and purple lesions: Erythematous candidosis.

Acute candidosis.

Chronic candidosis.

Denture-related stomatitis (denture sore mouth; chronic atrophiccandidosis).

Angular stomatitis (angular cheilitis; perleche).

Median rhomboid glossitis (central papillary atrophy of thetongue).

21 Red and purple lesions: Angiomas.


Venous lake (venous varix; senile hemangioma of lip).


22 Red and purple lesions: Proliferative vascular lesions,Kaposi sarcoma.

Proliferative vascular lesions.

Kaposi sarcoma.

23 Red and purple lesions: Erythroplakia.

Erythroplakia (erythroplasia).

24 Red and purple lesions: Erythema migrans (lingual erythemamigrans; benign migratory glossitis; geographical tongue;continental tongue).

25 Swellings: Hereditary conditions, drug-inducedswellings.

Hereditary gingival fibromatosis (HGF).

C1 esterase inhibitor deficiency (hereditary angioedema).

Drug-induced gingival swelling.

26 Swellings: Infections, human papilloma virus.


Warts (verrucae).

Multifocal epithelial hyperplasia (Heck disease).

Koilocytic dysplasia.

HPV and oral cancer.

27 Swellings: Granulomatous conditions.


Crohn disease and orofacial granulomatosis.

28 Swellings: Reactive lesions.

Denture-induced hyperplasia (epulis fissuratum).

Fibroepithelial polyp (fibrous lump).


Giant cell epulis (peripheral giant cell granuloma).

Pyogenic granuloma.

29 Swellings: Malignant neoplasms, oral squamous cellcarcinoma (OSCC).

30 Swellings: Malignant neoplasms, lymphoma, metastaticneoplasms.


Metastatic oral neoplasms.

31 Ulcers and erosions: Local causes, drug-inducedulcers.

Local causes.

Eosinophilic ulcer (traumatic eosinophilic granuloma; traumaticulcerative granulomatous disease).

Drug-induced ulcers (stomatitis medicamentosa).

32 Ulcers and erosions: Aphthae.

33 Ulcers and erosions: Aphthous-like ulcers.

Behcet syndrome (BS, Behcet disease).

34 Ulcers and erosions: Blood diseases, gastrointestinaldisorders.

Blood diseases.


Gastrointestinal disorders.

Celiac disease (gluten sensitive enteropathy).

35 Ulcers and erosions: Infections.

Hand, foot and mouth disease (HFM; vesicular stomatitis withexanthem).


Bacterial infections.

Acute necrotizing ulcerative gingivitis (Vincent disease; acuteulcerative gingivitis, AUG, ANG, ANUG).




36 Ulcers and erosions: Erythema multiforme, toxic epidermalnecrolysis and Stevens-Johnson syndrome.

Erythema multiforme.

Toxic epidermal necrolysis (TEN, Lyell syndrome) andStevens-Johnson syndrome (SJS).

37 White lesions: Candidosis (candidiasis).

Acute pseudomembranous candidosis.

Chronic hyperplastic candidosis (Candidal leukoplakia).

Chronic mucocutaneous candidosis (CMC).

38 White lesions: Keratosis, leukoplakia.

Tobacco-related keratosis.


39 White lesions: Hairy leukoplakia, lichen planus.

Hairy leukoplakia.

Lichen planus (LP) and lichenoid reactions.

40 Salivary conditions: Salivary swelling and salivaryexcess.

Salivary swelling.

Saliva excess (sialorrhea, hypersialia, hypersalivation,ptyalism) and drooling.

41 Salivary conditions: Dry mouth.

42 Salivary conditions: Sjogren syndrome.

43 Salivary conditions: Sialolithiasis, sialadenitis.



Sialadenitis: Acute viral (mumps).

Sialadenitis: Acute bacterial ascending.

Sialadenitis: Chronic bacterial.

Sialadenitis: Recurrent parotitis of childhood.

44 Salivary conditions: Neoplasms.

Benign neoplasms (adenomas).

Malignant neoplasms.

45 Salivary conditions: Mucoceles, sialosis.

Mucoceles (mucous cyst; mucus extravasation phenomenon; myxoidcyst).

Sialosis (sialadenosis).

46 Neck swelling.

Discrete swellings in the neck.

Cervical lymphadenopathy.

Unexplained lymphadenopathy.

Diffuse swelling of the neck.

47 Neck swelling: Cervical lymphadenopathy in generalizedlymphadenopathy.

Systemic infections.

Inflammatory disorders (not known to be infective).

Neoplastic causes.



48 Neurological conditions: Bell palsy, and trigeminalsensory loss.

Bell palsy.

Trigeminal sensory loss.

49 Neurological conditions and pain: Local, referred andvascular.

Local causes of orofacial pain.

Referred causes of orofacial pain.

Vascular causes of orofacial pain.

50 Neurological conditions and pain: Trigeminalneuralgia.

Trigeminal neuralgia.

51 Neurological conditions and pain: Psychogenic (idiopathicfacial pain, idiopathic odontalgia and burning mouth syndrome (oraldysesthesia)).

Persistent idiopathic, or unexplained (atypical) facial pain(IFP).

Burning mouth syndrome (BMS, glossopyrosis,glossodynia, oral dysesthesia, scalded mouth syndrome, orstomatodynia).

52 Jaw conditions: Temporomandibularpain-dysfunction.

Temporomandibular joint pain-dysfunction syndrome (TMPD),myofascial pain dysfunction (MFD), facial arthromyalgia (FAM),mandibular dysfunction, or mandibular stress syndrome.

53 Jaw bone conditions: Radiolucencies andradiopacities.



Mixed radiolucent and radiopaque lesions.

54 Jaw bone conditions: Odontogenic diseases andcysts.

Odontogenic infections.

Odontogenic cysts.

55 Jaw bone conditions: Odontogenic tumors.

Benign odontogenic tumors.

Malignant odontogenic tumors.

56 Jaw conditions: Bone disorders.

Non-neoplastic diseases.

Neoplastic disorders.

57 Jaw bone conditions: Fibro-osseous lesions.

Osseous dysplasia, cemento-osseous dysplasia (COD), periapicalcemental or cemento-osseous dysplasia (PCD).


Fibrous dysplasia.


Ossifying fibroma (cemento-ossifying fibroma).

Paget disease of bone.

58 Maxillary sinus conditions.

Rhinosinusitis (sinusitis).


59 Oral malodor.

60 Human immunodeficiency virus (HIV) infection andAIDS.


Product Details

  • publication date: 23/04/2010
  • ISBN13: 9781405199858
  • Format: Paperback
  • Number Of Pages: 128
  • ID: 9781405199858
  • weight: 384
  • ISBN10: 1405199857

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